Intra-amniotic administration of urinary trypsin inhibitor preserves intestinal contractility in meconium induced intestinal damage in chick embryos with gastroschisis.

Karakus, Osman; ATEŞ, OĞUZ; Murat, NERGİZ; HAKGÜDER, FAİKA; OLGUNER, MUSTAFA; Solmaz, Bora; ÖZER, ERDENER; Gidener, Sedef; Akgur, Feza

doi:10.1016/j.jpedsurg.2012.10.002

Intra-amniotic administration of urinary trypsin inhibitor preserves intestinal contractility in meconium induced intestinal damage in chick embryos with gastroschisis.

Atıf İçin Kopyala

Karakus O. Z., ATEŞ O., Murat N., HAKGÜDER F. G., OLGUNER M., Solmaz B., ...Daha Fazla

Journal of pediatric surgery, cilt.48, sa.7, ss.1495-8, 2013 (SCI-Expanded)

Yayın Türü: Makale / Tam Makale
Cilt numarası: 48 Sayı: 7
Basım Tarihi: 2013
Doi Numarası: 10.1016/j.jpedsurg.2012.10.002
Dergi Adı: Journal of pediatric surgery
Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
Sayfa Sayıları: ss.1495-8
Anahtar Kelimeler: Gastroschisis, Intestinal contractility, Intestinal damage, Urinary Trypsin Inhibitor, ALLANTOIC FLUID EXCHANGE, HUMAN NEONATAL URINE, AMNIOTIC-FLUID, SMOOTH-MUSCLE, RABBIT MODEL, FETAL URINE, BOWEL, PREVENTION, ETIOLOGY, INCREASES
Dokuz Eylül Üniversitesi Adresli: Evet

Özet

Background: Intestinal damage causes intestinal dysmotility in gastroschisis. Urinary trypsin inhibitor (UTI) has been shown to prevent intestinal damage in chick embryos with gastroschisis. The effect of intra-amniotic administration of UTI on intestinal motility in gastroschisis has not been investigated.