Lack of association between the Glu298Asp polymorphism of endothelial nitric oxide synthase and slow coronary flow in the Turkish population

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ÇAĞLAYAN A. O., KALAY N., Saatci C., Yalcin A., Akalin H., DÜNDAR M.

CANADIAN JOURNAL OF CARDIOLOGY, vol.25, no.3, 2009 (SCI-Expanded) identifier

  • Publication Type: Article / Article
  • Volume: 25 Issue: 3
  • Publication Date: 2009
  • Doi Number: 10.1016/s0828-282x(09)70044-3
  • Journal Name: CANADIAN JOURNAL OF CARDIOLOGY
  • Journal Indexes: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Keywords: Genetics, Receptor, Renin-angiotensin system, Slow coronary flow, ARTERY-DISEASE, GENE POLYMORPHISM, MYOCARDIAL-INFARCTION, VARIANT, ENOS, ATHEROSCLEROSIS, HYPERTENSION, DYSFUNCTION, PROTEINS, PATHWAY
  • Dokuz Eylül University Affiliated: No

Abstract

BACKGROUND: Coronary endothelial dysfunction plays an important pathogenetic role in patients with slow coronary flow (SCF). No data exist regarding the possible contribution of the Glu298Asp polymorphism genotype of the endothelial nitric oxide synthase (eNOS) gene to human SCF in the literature.