Lack of association between the Glu298Asp polymorphism of endothelial nitric oxide synthase and slow coronary flow in the Turkish population

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ÇAĞLAYAN A. O., KALAY N., Saatci C., Yalcin A., Akalin H., DÜNDAR M.

CANADIAN JOURNAL OF CARDIOLOGY, cilt.25, sa.3, 2009 (SCI-Expanded) identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 25 Sayı: 3
  • Basım Tarihi: 2009
  • Doi Numarası: 10.1016/s0828-282x(09)70044-3
  • Dergi Adı: CANADIAN JOURNAL OF CARDIOLOGY
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Anahtar Kelimeler: Genetics, Receptor, Renin-angiotensin system, Slow coronary flow, ARTERY-DISEASE, GENE POLYMORPHISM, MYOCARDIAL-INFARCTION, VARIANT, ENOS, ATHEROSCLEROSIS, HYPERTENSION, DYSFUNCTION, PROTEINS, PATHWAY
  • Dokuz Eylül Üniversitesi Adresli: Hayır

Özet

BACKGROUND: Coronary endothelial dysfunction plays an important pathogenetic role in patients with slow coronary flow (SCF). No data exist regarding the possible contribution of the Glu298Asp polymorphism genotype of the endothelial nitric oxide synthase (eNOS) gene to human SCF in the literature.