Balkan Radiology Congress 2024 , İzmir, Türkiye, 23 - 26 Ekim 2024, (Özet Bildiri)
INTRODUCTION
Left ventricular non-compaction describes the presence of excessive trabeculation of the left ventricle. The most common clinical presentations at the time of the first diagnosis are heart failure, thromboembolic events, and different rhythm disturbances.In our daily practice with the patients referred to our department for CAD investigation with coronary CT , we have detected left ventricular non-compaction without accompanying significant coronary artery stenosis. In this study, we aimed to investigate if non-compaction may mimic initial CAD symptoms.
METHODS
Between January 2021 and September 2024, coronary CTAs obtained in our department with suspicion of CAD were scanned and patients with coincidentally detected left ventricular non-compaction were collected. For the control group, patients who were referred with suspected CAD and underwent coronary CTA were selected randomly for a period of 3 months starting from January 2024, matched in terms of age, gender, and comorbidity, but not detected as non-compaction. Our patient group included 43 patients with non-compaction. The selected patient and control groups were evaluated in terms of CAD severity using the CAD-RADS 2.0 classification. Symptoms are noted as chest pain, atypical pain, palpitations, exertional dyspnea. Additionally, the noncompacted myocardium/compacted myocardium ratio was evaluated in the patient group.
FINDINGS
The increase in trabeculation observed in NC-CMP can be measured with CT. For this purpose, Melendez-Ramirez et al. accepted that a non-compact/compact myocardium ratio of 2.3 and above in at least 2 segments in a 17-segment heart model is diagnostic for NC-CMP.The highest NC/C ratio was noted in the non-compaction group and its average was 2,88. In our NC group, no significant coronary artery stenosis was detected in %90.7(CAD-RADS 0,1,2).In our control group, significant CAD stenosis is seen at the rate of %25.5 (CAD-RADS, 3,4,5).A statistically significant higher level of coronary artery disease was found in the control group between these two groups.
CONCLUSION
Although the non-compaction and control groups had similar presenting symptoms, age, gender and comorbidity factors, no significant CAD was detected in the NC group. This suggests that NC may present with symptoms mimicking CAD in the early period, before the typical late-term findings appear, as stated in our hypothesis.