V. Chelban Et Al. , "Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications," Nature communications , vol.15, no.1, pp.2269, 2024
Chelban, V. Et Al. 2024. Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications. Nature communications , vol.15, no.1 , 2269.
Chelban, V., Aksnes, H., Maroofian, R., LaMonica, L. C., Seabra, L., Siggervåg, A., ... Devic, P.(2024). Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications. Nature communications , vol.15, no.1, 2269.
Chelban, Viorica Et Al. "Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications," Nature communications , vol.15, no.1, 2269, 2024
Chelban, Viorica Et Al. "Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications." Nature communications , vol.15, no.1, pp.2269, 2024
Chelban, V. Et Al. (2024) . "Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications." Nature communications , vol.15, no.1, p.2269.
@article{article, author={Viorica Chelban Et Al. }, title={Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications}, journal={Nature communications}, year=2024, pages={2269} }